MCQ: Seizures & Skin lesions

Clinical scenario: Focal seizures | Skin lesions | Cortical tubers

A 2-year-old boy is referred for evaluation of recurrent focal seizures and developmental delay. His parents report that he has had several episodes of abnormal movements since infancy.

On examination:

  • Multiple hypopigmented macules are seen over the trunk.
  • A rough, thickened plaque is present over the lower back.
  • Multiple reddish papules are present over the cheeks.

MRI brain shows:

  • Multiple cortical tubers
  • Subependymal nodules along the lateral ventricles

Renal ultrasound demonstrates multiple bilateral renal lesions.

Which of the following is the most likely diagnosis?

A. Sturge-Weber syndrome
B. Neurofibromatosis type 1
C. Tuberous sclerosis complex
D. Von Hippel-Lindau disease
E. Ataxia-telangiectasia

Correct answer & Explanation:

Correct Answer: C. Tuberous sclerosis complex

Explanation:

This child has tuberous sclerosis complex (TSC), an autosomal dominant neurocutaneous disorder caused by mutations in:

  • TSC1 gene → hamartin
  • TSC2 gene → tuberin

These mutations cause activation of the mTOR pathway, resulting in multiple hamartomatous lesions.

Key clinical features:

Skin

  • Hypomelanotic macules (“ash leaf spots”)
  • Facial angiofibromas
  • Shagreen patch

Brain

  • Cortical tubers → seizures, developmental delay
  • Subependymal nodules
  • Subependymal giant cell astrocytoma (SEGA)

Kidney

  • Angiomyolipomas
  • Renal cysts

Why the other options are incorrect

  • A. Sturge-Weber syndrome: Port-wine stain in V1 distribution, leptomeningeal angioma, seizures, and tram-track cortical calcifications.
  • B. Neurofibromatosis type 1: Café-au-lait spots, neurofibromas, axillary freckling, and optic pathway gliomas.
  • C. Tuberous sclerosis complex: Correct — cortical tubers + hypopigmented macules + renal lesions.
  • D. Von Hippel-Lindau disease: Retinal angiomas, CNS hemangioblastomas, and renal cell carcinoma; does not cause cortical tubers.
  • E. Ataxia-telangiectasia: Progressive ataxia, telangiectasia, and immunodeficiency; no hamartomatous lesions.

Learning Point

Important neurocutaneous syndromes:

SyndromeSkin findingCNS finding
Tuberous sclerosisAsh leaf spots, angiofibromasCortical tubers, SEGA
Sturge-WeberPort-wine stain (V1)Leptomeningeal angioma, seizures
NF1Café-au-lait spotsNeurofibromas, optic glioma
NF2Few skin findingsBilateral vestibular schwannomas
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